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Therefore, it really is conceivable which the JIP1/3 → ASK1 → MKK4 signaling module might serve as a conduit to activate JNK when endosomal trafficking is disrupted and glucose concentrations drop through the early levels of methuosis. Our acquiring that MKK4 was robustly activated from the methuosis-inducing IPPs, MOMIPP and 2q, and le